![]() ![]() It can progress to an acute phase with more serious symptoms such as seizures, tremors, coma, and, even death. Effective treatment programs include corticosteroids and plasma exchange, suggesting pathologies in the blood plasma.Įncephalitis is an inflammation of the brain, often associated with infection, which appears initially with symptoms such as headache, fever, confusion, drowsiness, and fatigue. Spontaneous full recovery is the norm, although a recurrence can be provoked by vaccination. Characteristic symptoms include headache, fever, seizures and coma, distinguishing the condition from multiple sclerosis. ![]() It is estimated that up to 5% of the ADEM cases are post-vaccination encephalopathies. ADEM develops in approximately one in 1,000 measles cases, but can also occur less commonly following other viral infections such as varicella zoster and rubella. While it is usually associated with a viral infection, some cases are characterized by an autoimmune response without obvious infection, as is the case for anti- N-methyl-D-aspartate (NMDA) receptor encephalitis. However, others have been unable to detect an excess of such opiates in urinary analyses of children with ASD, and this may suggest that some other factor is involved.Īcute disseminated encephalomyelitis (ADEM) is an inflammatory demyelinating disorder of the central nervous system, which can also be provoked by vaccination, and occurs most often in children, especially infants. It is argued that incomplete digestion of certain opiogenic peptides such as the exorphin, β-caseomorphine, followed by their penetration into the brain, can modulate the GABA-ergic, serotoninergic, dopaminergic, and noradrenergic systems. In ASD and hepatic disease, a leaky gut and/or impaired liver function and an impaired blood brain barrier result in the penetration of both allergenic peptides and toxins produced by gut bacteria into the blood stream and the brain, causing neurological effects. A compelling hypothesis for the etiology of the autism spectrum, based on the concept of “entero-colonic encephalopathy”, is developed in, where parallels are drawn with hepatic encephalopathy associated with liver failure. ![]() It is now well established that such disorders, especially the more severe types at the center of the spectrum, are associated with gastrointestinal problems in addition to the neurological impairment. Given these facts, dietary and lifestyle changes, including increased sulfur ingestion, organic whole foods, increased sun exposure, and avoidance of toxins such as aluminum, mercury, and lead, may help to alleviate symptoms or, in some instances, to prevent autism altogether.Īutism spectrum disorders (ASD), with early childhood autism at their core, are loosely defined by social, cognitive, and memory deficits leading to atypical neurodevelopment. Several environmental factors can synergistically promote the encephalopathy of autism, including the herbicide, glyphosate, aluminum, mercury, lead, nutritional deficiencies in thiamine and zinc, and yeast overgrowth due to excess dietary sugar. Research is cited showing how taurine may not only help protect neurons from hypochlorite exposure, but also provide a source for sulfate renewal. Encephalitis, while life-threatening, can result in partial renewal of sulfate supply, promoting neuronal survival. We argue that impaired synthesis of cholesterol sulfate in the skin and red blood cells, catalyzed by sunlight and nitric oxide synthase enzymes, creates a state of colloidal instability in the blood manifested as a low zeta potential and increased interfacial stress. This paper makes two claims: (1) autism can be characterized as a chronic low-grade encephalopathy, associated with excess exposure to nitric oxide, ammonia and glutamate in the central nervous system, which leads to hippocampal pathologies and resulting cognitive impairment, and (2), encephalitis is provoked by a systemic deficiency in sulfate, but associated seizures and fever support sulfate restoration. ![]()
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